STEM Graduates is a graduate recruitment agency and jobs board. We offer permanent salaried roles to students and graduates from Science, Technology, Engineering and Mathematics disciplines. We believe these candidates have a unique set of career needs that can only be met by a specialist within this field. We launched STEM Women in 2016 to provide a specific place for female careers advice, profiles of women in STEM and a dedicated job board.
We are always looking to expand what we can offer STEM students to make them more employable in their highly competitive markets. That is why we are excited to launch our recent partnership with the Science Council. This partnership will include a variety of activities and exciting content.
The Science Council will host a careers advice blog topic each month written by STEM Graduates (most recent blog post here) and we will be focusing on educating our candidates on the benefits on offer from joining the Science Council. We will also be listing the numerous specific professional bodies under the science umbrella including the Royal Society of Biology, the Institute of Science and Technology and the Institution of Environmental Sciences. We will achieve this through our social media channels, a new dedicated section on our website and with weekly articles.
We are proud to endorse the ‘working towards registered scientist’ (Registered Scientist (RSci)) initiative that has been launched by the Science Council. This will focus on the conduct, competence and professional development of early years’ scientists. For many graduates this initiative will be the first step towards becoming a chartered scientist.
The Science Council is a membership organisation for professional bodies and learned societies across the disciplines of science. They are in a unique position, bringing together a range of disciplines and sectors to reflect the multi-disciplinary practice of science in today’s society.
We are looking to expand this section further with other societies, associations and communities so please let us know if you have any ideas.
View of the head of a transparent 7 week embryo labelled with Peripherin. Cranial nerves have been pseudocoloured. From Fig1. Belle et al., Cell, 2017
Throughout history, the desire of scientists to understand physiology and disease by thoroughly studying anatomical features, has always faced an intractable limitation: they cannot simply see through the tissue! Dissection has therefore been the modus operandi of anatomists: from Galen’s pioneering studies, to modern day biologists who routinely section tissues to label structures for microscopic analysis.
Whilst these methods have informed a wealth of knowledge linking anatomical form to function, they are inherently flawed due to a 3-Dimensional appreciation of structures being lost. This has been especially problematic for the study of human development, where structures are continually evolving, and therefore a precise visualisation has been impossible to achieve through traditional methods and anatomical atlases. Coupled with the difficulties in tissue access, our understanding of human development has progressed perhaps the slowest of any biological process since the 1930’s; whilst in some cases observations of lower vertebrates have subsequently been erroneously applied to humans.
“Birth defects of structural or functional origin currently affect more than 3% of births”
Without a good understanding of physiological development, we lack the fundamental knowledge required for clinicians and researchers to tackle a healthcare issue that inflicts a severe healthcare and emotional burden. Recent advances in non-invasive, in vivo imaging techniques, have shown great promise in detecting congenital abnormalities as well as providing information on gross topological features of fetal development; however, they lack sufficient resolution in order to inform developmental biologists of currently unknown features of organogenesis. This has recently been most strikingly highlighted by the surge in Zika virus infections and reports of its detrimental effects on cephalic development.
The developing innervation of the human hand from 7-11 weeks gestation. From Fig2. Belle et al., Cell, 2017
In order to begin rapidly addressing the gaping holes in our knowledge, we have begun to form a cutting edge research consortium of developmental biologists, to build a detailed human cell atlas. Here we have expanded the use of clearing techniques to human tissues and immunolabeled with over 40 antibodies, intact human embryos during the first trimester of gestation (from 6 to 14 gestational weeks).
In recent years, several techniques have been developed for tissue clearing, in which whole organs are rendered macromolecule permeable and optically transparent. Among tissue-clearing techniques, the process called 3D imaging of solvent-cleared organs, or 3DISCO has been proved to be a simple, robust and inexpensive method for 3D analysis of immunolabeled transparent organs in embryonic and postnatal mice.
Currently organised along seven organ systems, the project aims to expand and evolve as more data is added. Current data in the atlas examines molecular organogenesis based on over 40 samples comprising 1,500,000 optical sections – making it the most complete 3D analysis of early human development currently available.
Constructed by imaging intact tissues and whole embryos, the atlas will be an invaluable tool for researchers with the ability to explore cell distributions, count proliferating cells in each organ etc. whilst also being useful for didactic purposes, with the ability to 3D print models to inform health science teaching programs.
This has long been the dream of developmental biologists, which has finally been realised by the use of organic solvent based clearing techniques (3DISCO/iDISCO) combined with light sheet imaging. Together, these powerful approaches allow inexpensive labelling of any cell population of all developing organ systems during development and imaging at cellular resolution whilst fully maintaining structural relationships. To further highlight the robustness and power of this technique to analyse human development, it should be noted that these data were generated in just over one year by a small team of researchers.
And this is just the beginning, as the online resources are open access – available for researchers all over the world to analyse and contribute to, along with new data we will acquire. The goal of the project is to make a continuously updated 3D molecular reference atlas of human cells during development, paramount to a better understanding of human development during health and disease.
The online video series of immunolabeled tissues along with the original data sets is available at https://transparent-human-embryo.com/ . The 3D database was developed with Keen eye technologies with support from the “fondation voir et entrendre”
University of Oregon biologists have figured out how zebrafish perfectly regenerate amputated fins with a precisely organized skeleton.
Adult zebrafish fins, including their complex skeleton, regenerate exactly to their original form within two weeks after an amputation. The process, they found, is driven by clusters of specialized skin cells that migrate over reforming bones, known as rays, and escort bone cells into the right positions to form individual bones of a branched skeleton.
These skin cells produce a protein called Sonic hedgehog, which interacts with bone-building cells called osteoblasts to promote bone patterning during fin regeneration.
“The orderly reconstruction of zebrafish fins is amazing to see,” said Kryn Stankunas, a professor in the Department of Biology and member of the Institute of Molecular Biology. “Zebrafish fins, which are akin to our limbs, regenerate perfectly. The zebrafish bony rays re-branch just like the original structure. This would be like losing your arm and watching it progressively regenerate complete with a hand and fingers — all the bones restored in their original configuration.”
The findings will not lead to humans re-growing lost limbs, Stankunas said, but such advances in understanding the fundamental processes of regeneration in related vertebrate organisms will inform innovative and targeted therapeutic strategies to improve the repair of broken bones.
“The mechanism — how the skin and bone cells dynamically move and interact using the signaling pathway — is elegant and unexpected, broadening the project’s impact on regenerative medicine,” Stankunas said.
Hedgehog signaling, he added, is also linked to several human cancers.
“The zebrafish fin provides a tractable and simple model to decipher mechanisms of regenerative skeletal patterning,” the researchers wrote in their paper in the March 28 issue of the journal Development, a publication of the non-profit Company of Biologists in the United Kingdom.
Benjamin E. Armstrong, who earned a doctorate in biochemistry in 2016, was the study’s lead author. Scott Stewart, a research professor in the Institute of Molecular Biology, co-directed the project.
Green fluorescent proteins show where bone-building is occurring in the regeneration of a zebrafish caudal fin that had been amputated. Complete repairs begin at the tail’s base and gradually proceed to the tip, a process that is completed within two weeks. Courtesy of Kryn Stankunas
The research team used genetically modified zebrafish that produces a fluorescent protein that helps identify the subset of skin and bone cells that respond to Hedgehog signals. The fluorescent marker appears green under the microscope until illuminated with ultraviolet light to photo-convert the green protein to red.
This photo-conversion method revealed that repairing skin cells collectively move towards the tip of the regenerating fin. At particular times, Sonic hedgehog is induced in skin cell clusters that then split into two pools. Simultaneously, the skin cells activate a Hedgehog response in adjacent osteoblasts. That drives them to associate with the skin cells and co-migrate into split groups. The now separated bone cells continue to regenerate replacement bone, but now forming two rays instead of one – a branched skeleton.
“We could see that the bone cells responding to the skin-produced Sonic hedgehog become physically attached to the migrating skin cells”
“We could see that the bone cells responding to the skin-produced Sonic hedgehog become physically attached to the migrating skin cells,” Stewart said. “The pathway is quickly turned off but the now split groups of bone cells will then form two separated mature bony rays connected at a branch point.”
To define the functions of the Hedgehog signaling pathway, the researchers used a new chemical inhibitor, BMS-833923, to turn off Hedgehog signaling in their experimental fish. With Hedgehog blocked, the skin and bone cells failed to interact, and the fin regenerated with stick-like rays rather than forming a branched skeleton.
The inhibitor used in the study is in clinical trials against some forms of human cancers, but it had not been used in zebrafish. The Hedgehog pathway is most associated with basal cell carcinoma and medulloblastoma, Stankunas said.
“The Hedgehog response is absolutely required for branching and not essential for any other aspect of regeneration,” Stankunas said. “Instructions that drive the branching come from the skin cells moving into two groups and likewise dividing the osteoblasts. This is new information. It is the traffic pattern generated by the signaling that regenerates the fin. It is skin and bone working together.”
###
Astra Henner, lab manager and research assistant, was the fourth co-author of the paper.
The National Institutes of Health funded the project through a training grant to Armstrong and research grants to Stankunas and Stewart.
Source: Kryn Stankunas, associate professor of biology, 541-346-7416, kryn@uoregon.edu
Note: The UO is equipped with an on-campus television studio with a point-of-origin Vyvx connection, which provides broadcast-quality video to networks worldwide via fiber optic network. There also is video access to satellite uplink and audio access to an ISDN codec for broadcast-quality radio interviews.
We are seeking an enthusiastic, highly motivated and productive postdoctoral research associate to join a BBSRC-funded research project investigating the formation of primordial follicles in the developing mammalian ovary, led by Dr Andrew Childs, Lecturer in the Department of Comparative Biomedical Sciences (Royal Veterinary College, University of London, UK).
The successful candidate will use a combination of in vitro and in vivo techniques to investigate how growth factor signalling converges with the transcriptional machinery in fetal ovarian somatic cells to regulate the timing and extent of follicle assembly in the mammalian ovary. The post-holder will be an active member of the research team, contributing to experimental design, data collection, analysis and dissemination, and student supervision.
The successful candidate should have a PhD in reproductive or developmental biology (or a closely related discipline). They will be proficient in a range of research techniques, including molecular biology, immunohistochemistry, and cell or organ culture. Excellent organisational and communication skills are essential, as is the ability to work independently and as part of a team. Experience of Chromatin Immunoprecipitation (ChIP), large scale transcriptomic analyses, and/or working with animal models would be advantageous.
Applicants should be available to start no later than 1st May 2017.
Prospective applicants are encouraged to contact Dr Andrew Childs (Lecturer, Comparative Biomedical Sciences) at achilds@rvc.ac.uk.
Here are the highlights from the new issue of Development:
Making thalamic neurons in vitro
In recent years, methods to derive multiple differentiated neuronal types from embryonic stem cells (ESCs) in vitro have been reported. Three-dimensional (3D) culture methods not only support differentiation but also recapitulate spatial aspects of brain development.
Such studies were pioneered by the late Yoshiki Sasai, and on p. 1211, his colleagues Atsushi Shiraishi and Keiko Muguruma adapt the original 3D culture conditions – which supported rostral neural fate – to derive thalamic neurons from mouse ESCs for the first time. They find that addition of insulin and FGF pathway inhibitors can specify caudal forebrain identity, and that subsequent treatment with BMP7 can promote thalamic fate. Within the neuroepithelial-sphere structure that forms in these cultures, there is significant spatial organisation: early progenitors are found by the apical cavity, while more mature cell types are located towards the outside, and the spheres display rostral-caudal regionalisation. The derived neurons can extend axons that – both in culture and in transplantation experiments in vivo – show projection patterns consistent with thalamic identity. Not only does this work allow the generation of thalamic neurons in vitro, but it also provides insights into the signalling mechanisms regulating thalamus development in vivo.
Bone regeneration in the fish fin
Zebrafish can fully regenerate their fins, a process that involves the reconstitution and patterning of multiple tissue types. New bone is regenerated via the de-differentiation, proliferation and re-differentiation of osteoblasts, which occur in a spatially organised manner to recapitulate the original fin shape and skeleton. How skeletal patterning – including outgrowth and bifurcation of new rays – is controlled in this context is incompletely understood, though is thought to involve Hedgehog signalling.
Kryn Stankunas, Scott Stewart and colleagues (p. 1165) now define distinct roles for the two Hedgehog ligands expressed in the regenerating fin: shha and ihha. shha is expressed in epidermal cells immediately adjacent to osteoblasts at the site of ray branching, and is required for branching. Intriguingly, it appears to act at short range, through direct contact with osteoblast progenitors via cellular protrusions, to promote splitting of the ray through cell movements. ihha, on the other hand, is expressed in the osteoblasts, where it promotes differentiation via a non-canonical signalling route. These data clarify the role of Hedgehog signalling in ray regeneration and shed light onto the mechanisms underlying skeletal patterning in regenerative contexts.
Turning off translation in germ cells
Stem cell quiescence has been reported in many systems, and typically involves the slowing or stalling of the cell cycle and low transcriptional activity. Primordial germ cells (PGCs) of sea urchin are known to enter a quiescent state prior to gastrulation, before re-activating later in development.
Now (p. 1201), Gary Wessel and co-workers show that this quiescence also involves a significant reduction in translational activity. Two potential mechanisms are uncovered. Firstly, Nanos2, which is expressed specifically in PGCs, binds to and downregulates the critical translation factor eIF1A. Secondly, mitochondrial number and activity is low in PGCs, which might induce a switch to glycolytic metabolism and hence an acidification of the cytoplasm. Increasing cellular pH promotes translational activity specifically in PGCs. This work raises many intriguing questions. For example, how is translational activity re-activated at later stages? How are the metabolic changes in PGCs orchestrated? How general might this be in quiescent stem cell populations? Thus, the identification of this previously unrecognised phenomenon of transient translational quiescence in sea urchin PGCs opens up many new avenues for investigation.
Timing is key to turn root into shoot
Plant cells show remarkable plasticity. For example, lateral roots can be converted into shoots by supplementing the culture medium with cytokinin, which induces shoot fate. When properly controlled, this conversion does not involve callus formation, and so allows a detailed analysis of the processes directing the switch of organ identity.
Using this system, Philippe Rech and colleagues (p. 1187) find that competence for root-to-shoot conversion is restricted to a narrow time window of lateral root development, coinciding with the stage at which the stem cell niche is formed in the new root. Furthermore, conversion can be reversed during this period – auxin treatment can switch the tissue back to a root – confirming that organ identity is not immediately fixed. Importantly, the authors provide evidence that root-to-shoot conversion does not occur via dedifferentiation, but rather via a direct transdifferentiation process. Transcriptome and methylome profiling provide insights into the gene expression and epigenetic changes occurring during conversion. This atypical mode of organogenesis may lead to novel methods for the vegetative multiplication of valuable plant cultivars.
How cell-cell contact defines fate
In many systems, stem cell fate is regulated by Notch signalling. One such example is the Drosophila midgut, where intestinal stem cells (ISCs) can divide either asymmetrically, generating a Notch-positive enteroblast (EB) and a Notch-negative ISC, or symmetrically, either forming two EBs or two ISCs. But what determines the outcome of ISC division, and how does Notch signalling influence this?
Joaquín de Navascués, Jordi Garcia-Ojalvo and co-workers address this question on p. 1177 using a combination of experimental and modelling approaches. Their key insight is that contact area between the two daughter cells correlates with cell fate: where the contact area is small, both cells tend to remain ISCs, where it is larger, one or both cells differentiate. Since Delta-Notch signalling involves direct contact between the two cells, the area of contact can influence the effective signalling threshold. Both the computational and experimental analyses support the idea that the pattern of cell fates following ISC division can be at least partly explained by variability in cell contact area, and hence in the levels of Notch-Delta signalling between the two daughter cells. That such a model might also apply in other stem cell systems is an intriguing possibility.
Spindle orientation: a question of complex positioning
Dan Bergstralh and colleagues discuss key features of the spindle-orientating complex and reviews how this complex is regulated and localized to allow correct mitotic spindle orientation.
Using synthetic biology to explore principles of development
Jamie Davies explores how synthetic biology-based approaches have been used to explore the principles underlying patterning, differentiation and morphogenesis during development.
Featured movie
In our featured movie, Naoto Ueno, Makoto Suzuki and colleagues show how two patterns of calcium fluctuation in the Xenopus neural plate control epithelial folding, with extracellular ATP and N-cadherin also participating in calcium-induced apical constriction.
Two years postdoctoral position at INSERM U1065 (C3M)-team 3, Nice, France
on the study of cell death of motor neurons. Starting ASAP.
A two-year postdoctoral position starting ASAP, funded by the French National Research Agency is available in the ‘Metabolic control of cell death’ team (INSERM U1065), located at the Archet Hospital in Nice, south of France.
Title: How mitochondrial dysfunction leads to motor neuron disease?
Recently, in close collaboration with Pr. Paquis-Flucklinger, we showed that mitochondrial dysfunction can have a causative effect in motor neuron degeneration. We reported a large family with a mitochondrial myopathy associated with motor neuron disease and cognitive decline looking like frontotemporal dementia (FTD). We identified a missense mutation (p.Ser59Leu) in the HCHD10 gene coding for a mitochondrial protein whose function was unknown (Genin EC et al. EMBO Mol Med 2015 Dec 14:58-72).
We and others reported CHCHD10 mutations in patients with dementia-amyotrophic lateral sclerosis (FTDALS) and familial or sporadic pure ALS.
Project: Amyotrophic lateral sclerosis is a devastating disease affecting upper and lower motor neurons leading to progressive failure of the neuromuscular system and death from respiratory failure. Among all factors involved in ALS pathogenesis, mitochondrial dysfunction has always been recognized as a candidate major player. However, whether mitochondria have a causative role in ALS has been always debated. Our results open a new field to explore the pathogenesis of motor neuron disease by showing that mitochondrial dysfunction may be at the origin of some of these phenotypes.
Our goals are:
(i) to better characterize the role of the CHCHD10 protein on cell death and to compare the effects of
different CHCHD10 mutations leading to different clinical phenotypes,
(ii) to understand how CHCHD10 mutations lead to motor neuron cell death by generating specific human cellular (IPS) and characterizing in vivo models,
Candidate profile:
The candidate should hold a PhD in physiology, pharmacology or related disciplines and have previous expertise in cell culture / characterization of primary neuronal cells.
Practice or knowledge of in vivo animal experimentation techniques as well as in cellular and molecular biology techniques would be appreciated.
How to apply?
Candidates should send a curriculum vitae with publication list, a short summary of research achievements, and the names and email addresses of at least two references to ricci@unice.fr
Dr. J-E Ricci
INSERM U1065, C3M
Directeur de l'équipe- 3
Batiment Universitaire Archimed
151 Route de Ginestière
BP 2 3194
06204 NICE Cedex 3
Tel 33+ (0)4 89 06 43 04
Fax 33+(0)4 89 06 42 21
Email: ricci@unice.fr
The Department of Neurology at the University of California, Irvine anticipates an opening for an Assistant Project Scientist in the Translational Laboratory and Biorepository (TLaB). This position requires solid background in experimental design and fluency in the use of technology germane to investigations of exosomes, including but not limited to, in vitro and in vivo methods for investigating extracellular vesicles, nanoparticle analysis, fluorescent sorting methods, immunoprecipitation, ultracentrifugation, and electron microscopy. Research efforts will involve quantitative analyses of exosomal cargo proteins and nucleic acids that are relevant in neurological disorders. The incumbent will be exposed to a wide variety of ongoing research studies within the TLaB related to neurodegenerative diseases, traumatic brain injury, and autism.
Requirements:
Candidates must hold a doctoral degree or equivalent in Neurobiological Sciences, Biochemistry, Bioengineering, Molecular or Cell Biology, or Protein and/or Nucleic Acid Chemistry and have a strong research background. Significant and creative contributions to a research or creative project in the field of Neurology and command of the English language (spoken and written) are expected. Preference will be given to candidates who hold a strong publication record and have post-doctoral experience and grant writing experience and prior funding.
Additional Information:
Positions are dependent upon extramural funding. Rank will be determined based on qualifications and experience. Initial appointments are for one year and renewal is based on continued availability of support. Salary will be commensurate with qualifications and experience.
Culture:
The UCI TLaB was established in 2015, with the research faculty and senior staff transferring from the Biomarker Laboratory and Biorepository at Georgetown University and the University of Rochester. Long-standing national and international collaborations exist on the primary research topics and provide opportunity for professional growth and job satisfaction. Current faculty and staff collaborate on a variety of human blood-based biomarker investigations, as well as in vitro and in vivo models related neurological disorders with a high potential for human translation.
The TLaB members are also involved in teaching and mentoring of medical students, graduate and undergraduate students.
Substantive inquiries about the position should be directed to:
Massimo S. Fiandaca, MD
Associate Professor
Department of Neurology
Co-Director, Translational Laboratory and Biorepository mfiandac@uci.edu
Office phone – 949-824-5579
Applicants should complete an online application profile and upload the following application materials electronically to be considered for the position:
Cover letter—Please discuss current research and future plans.
Curriculum vitae
Names and Contact Information of Three References
Diversity Statement
The University of California, Irvine is an Equal Opportunity/Affirmative Action Employer advancing inclusive excellence. All qualified applicants will receive consideration for employment without regard to race, color, religion, sex, sexual orientation, gender identity, national origin, disability, age, protected veteran status, or other protected categories covered by the UC nondiscrimination policy.
A postdoctoral fellow position is available in the laboratory of Dr. Paul Burridge in the Department of Pharmacology and the Center for Pharmacogenomics at Northwestern University Feinberg School of Medicine, Chicago, IL.
Successful candidates will participate in NIH- andAHA-funded projects to study the application of human induced pluripotent stem cells (hiPSCs) in predictive medicine. Our goal is to develop the next generation of tools for predicting drug responses and validating SNPs to allow the use of genomic information in precision medicine and drug discovery. With these data, we will probe the mechanisms of action of a range of drugs to provide individualized treatment selections and regimens to improve drug efficacy and eliminate off-target toxicity.
We are looking for highly motivated and creative candidates with an interest in studying SNPs and molecular mechanisms involved in chemotherapy-induced toxicity (primarily cardiovascular) of tyrosine kinase inhibitors and monoclonal antibodies using patient-specific hiPSC-derived cells. Projects will utilize a wide range of state-of-the-art techniques such as genome editing, high-content imaging, high-throughput drug screening, electrophysiology, whole genome sequencing,RNA-seq, and eQTL. The Burridge Laboratory is stably supported by NIH,AHA, and institutional funding. More about the lab can be found here:http://burridgelab.com/
Qualifications: PhD or MD/PhD degree (either about to graduate or graduated within the last year) and a strong record of peer-reviewed publications including first author publications are essential. Expertise in several of the following areas is required: mechanisms of chemotherapy agents and toxicity, disease modeling, pharmacogenomics,WGS,RNA-seq, eQTL,GWAS, bioinformatics,CRISPR-based genome editing, high-throughput biology and drug screening, electrophysiology, hiPSC derivation, culture, and differentiation (cardiac/vascular smooth muscle/endothelial/blood/hepatic/renal/neural), direct reprogramming, engraftment, and developmental/cardiovascular biology. Good verbal and written communication skills in English are essential. The successful candidate will join a dynamic research environment in the Department of Pharmacology, which offers both basic science and clinical translational opportunities to explore fundamental questions in pharmacogenomics.
Salary will be per theNIH(NRSA) Scale and commensurate with experience.
This position is highly suitable for those interested in pursuing a career as an independent academic scientist. Those without experience in the above fields, those more than one year after graduating their PhD, and those who wish to pursue a career in the pharma/biotechnology industry are asked not to apply.
Please send a CV, a cover letter containing a brief description of research experience and interests, and a list of 2-3 references to:paul.burridge@northwestern.edu
Northwestern University is an Equal Opportunity, Affirmative Action Employer of all protected classes, including veterans and individuals with disabilities. Women and minorities are encouraged to apply. Hiring is contingent upon eligibility to work in the United States.
I am delighted to announce that we are offering an opportunity for a 3-month internship on the Node. This is being offered as a PIPS placement to students on the BBSRC DTP program, or on similar programs where an internship forms part of the PhD training.
If you have a passion for science communication and writing, as well as a love of developmental biology, this could be the perfect internship for you! We will provide a great insight into what it’s like to work in the online scicomm environment, giving you the opportunity to come up with ideas for Node posts, talk to potential authors about writing for us, help Node users with their posts, and run the Node’s social media accounts. Working in a publishing company, you’ll also learn about how science publishing works from the inside.
You can find out more here, or please get in touch with me, Katherine Brown (Development’s Executive Editor) if you want to know more.
The Company of Biologists and its journal Development are looking for an intern, through the BBSRC DTP/PIPS or equivalent schemes, to help run the successful community website ‘the Node’ . This is a great opportunity to gain experience in the rapidly growing online science communication environment, to develop writing skills, and to learn about academic publishing.
Launched in 2010, the Node is the place for the developmental biology community to share news, discuss issues relevant to the field and read about the latest research and events. The intern will be involved in the day-to-day running of the Node, mentored by the Node’s Community Manager. The internship will be based in our office in Cambridge.
Core responsibilities of the position include:
Creating and commissioning content for the Node, including writing posts and soliciting content from the academic community, societies and other organisations
Providing user support
Running Development’s and the Node’s social media accounts (Twitter and Facebook)
The successful intern will have:
Relevant scientific expertise (ideally in developmental biology or a related field)
Strong writing and communication skills
Keen interest in science communication
Experience of and interest in blogging and/or social media (ideally including experience with WordPress)
The Company of Biologists (http://www.biologists.com) exists to support biologists and inspire advances in biology. At the heart of what we do are our five specialist journals – Development, Journal of Cell Science, Journal of Experimental Biology, Disease Models & Mechanisms and Biology Open – two of them fully open access. All are edited by expert researchers in the field, and all articles are subjected to rigorous peer review. We take great pride in the experience of our editorial team and the quality of the work we publish. We believe that the profits from publishing the hard work of biologists should support scientific discovery and help develop future scientists. Our grants help support societies, meetings and individuals. Our workshops and meetings give the opportunity to network and collaborate.
We are looking for an intern to start in Autumn 2017, though can be somewhat flexible with start dates and encourage interested candidates to submit their application as soon as possible. To apply, please send a CV and cover letter, stating why you are interested in this opportunity, to recruitment@biologists.com and Katherine Brown (Development’s Executive Editor) at katherine.brown@biologists.com. Please also direct informal enquiries to the same addresses.
(4 votes)
Loading...
(3 votes)
the University of Oregon 
(No Ratings Yet)
Now (p. 
